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Thereby, the heart, lung, liver, and kidney sustain major impacts and have high MDA values that affect these organ lesions and vice versa and, once counteracted, indicate the value of the therapy effect

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In the endogenous apoptotic pathway, various cellular stress, such as nuclear DNA damage, cytokine deprivation, endoplasmic reticulum stress (ERs), lysosomal permeabilization, and oxidative stress generate intracellular death signals to promote mitochondrial dysfunction and subsequently mitochondrial outer membrane pore formation (MOMP)