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These effects were accompanied by enhanced hepatic fatty acid catabolism and a reduction in the tissue n-6/n-3 PUFA ratio, underscoring the role of n-3 PUFAs in counteracting chronic TNFα-driven inflammation and metabolic dysfunction ( Beyond general inflammation, EPA has also demonstrated a targeted ability to inhibit specific pro-tumorigenic pathways
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Advances of ferroptosis pathways in acute myeloid leukemia
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