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Although one cannot exclude toxic effects due to Plk4 over-expression, the consequential inhibition of cell proliferation or apoptosis has been directly linked to centrosome amplification 6 , whereas non-centrosomal PLK4 functions reported to date are predominantly pro-survival or pro-invasive 19

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Glucagon-like peptide-1 receptor regulates endoplasmic reticulum stress-induced apoptosis and the associated inflammatory response in chondrocytes and the progression of osteoarthritis in rat

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