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In the brains of children with ASD, low levels of mitochondrial glutathione and mitochondrial dysfunction have been reported (24, 26, 52, 53)

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4 Modeling core AD pathologies with brain organoids 4.1 Extracellular deposition and aggregation of amyloid-β plaques Organoid technology has brought a new level of research capability to AD studies because it allows the investigation of core disease mechanisms that involve Aβ plaque development and Aβ accumulation in cellular membranes (Table 1)