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The interplay between pharmacokinetic genes—notably CYP2C19 and CYP2D6 —and pharmacodynamic genes such as SLC6A4 (the serotonin transporter) and HTR2A (the serotonin 2A receptor) provides a comprehensive framework to explain the wide heterogeneity observed in patient responses to selective serotonin reuptake inhibitors (SSRIs)
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