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Moreover, intracerebroventricular infusion of recombinant rat CX3CL1 enhanced SE-induced neuronal damage, which, however, was largely prevented by the infusion of antibodies of CX3CL1 or CX3CR1 [129], suggesting that the CX3CL1/CX3CR1 axis may contribute to SE-induced neuronal damages via neuron-glia interactions
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