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By inhibiting NNMT: NAD+ metabolism is modulated, potentially improving mitochondrial function Adipogenesis (fat cell formation) may be reduced Muscle cell metabolism may be improved Animal studies suggest fat loss without muscle wasting This is distinct from NAD+ precursors like NMN or NR — 5-Amino-1MQ works upstream on NNMT rather than supplementing NAD+ directly

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The prodrug was stable in normal tissues but can be easily hydrolyzed into DON in P493B lymphoma cells, inhibiting tumor cell proliferation in a dose-dependent manner (Tenora et al., 2019)