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Heat shock proteins have protective roles in the gut epithelial cells and their expression is mediated by commensal bacteria through the ligation of the bacterial lipopolysaccharide (LPS) with the host toll-like receptor 4 (TLR4) [88]

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Therefore, in most cases, the new data are unable to establish statistically significant changes from the status quo, especially since the old PE/HR measures were themselves estimated with substantial levels of statistical uncertainty